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June 01, 2017; 129 (22)

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Histological analysis of bone marrow from an Apcdel/+ Ctnnb1del/+ mouse is similar to that from a wild-type mouse, illustrating that a decrease in Ctnnb1 levels and Wnt signaling reverses the myelodysplastic syndrome phenotype observed in Apcdel/+ mice. See the article by Stoddart et al on page 2959.