Lane SW, Sykes SM, Al-Shahrour F, et al. The Apcmin mouse has altered hematopoietic stem cell function and provides a model for MPD/MDS. Blood. 2010;115(17):3489-3497.

On page 3495 in the 29 April 2010 issue, Figure 5C is an inadvertent replication of supplemental Figure 9. The figure legend references within text are correct. The authors do not believe this replication compromises the quality of data or message of the article. The corrected Figure 5 is shown below.

Figure 5

Apcmin MDS/MPD is predominantly cell extrinsic. (A) Transplantation of 106 bone marrow cells from Apcmin MDS/MPD into sublethally irradiated CD45.1 recipients. White cell count (WCC × 103/μL), hematocrit (HCT%), and donor cell chimerism (%)16 weeks after transplantation. WCC: 9.9 × 103/μL Apcmin versus 11.5 × 103/μL WT, P = .12; HCT: 42.5% Apcmin versus 42.9% WT, P = .81; 51.4% Apcmin versus 53.4% WT, P = .75. (B) Bone marrow transplantation of 1 × 106 CD45.1 congenic bone marrow cells into sublethally irradiated Apcmin or WT recipients. Sublethal irradiation was used because of previous reports describing intolerance of lethal irradiation in Apcmin mice.5 WCC × 103/μL, HCT%, spleen weight (g) WCC: 8.64 × 103/μL Apcmin versus 7.53 × 103/μL WT, P = .59; HCT: 17.3% Apcmin versus 31.8% WT, P = .01; spleen: 0.41 g Apcmin versus 0.11 g WT, P < .01. (C) Peripheral blood parameters, including red cell count (RCC × 106/μL), hematocrit (HCT%), mean corpuscular volume (MCV fl), mean corpuscular hemoglobin (pg), red cell distribution width (RDW%), and platelet count (×103/μL).