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The BCL6 transcriptional program features repression of multiple oncogenes in primary B cells and is deregulated in DLBCL

Weimin Ci, Jose M. Polo, Leandro Cerchietti, Rita Shaknovich, Ling Wang, Shao Ning Yang, Kenny Ye, Pedro Farinha, Douglas E. Horsman, Randy D. Gascoyne, Olivier Elemento and Ari Melnick

Article Information

Citation 
vol. 113 no. 22 5536-5548
PubMed 

Print ISSN 
Online ISSN 
History 
  • Submitted December 29, 2008
  • Accepted March 17, 2009
  • Published online May 28, 2009.

Article Versions


Contributors 
  • Weimin Ci, 1Division of Hematology/Oncology, Department of Medicine, Weill Cornell Medical College, New York, NY;
  • Jose M. Polo, 2Department of Developmental and Molecular Biology and
  • Leandro Cerchietti, 1Division of Hematology/Oncology, Department of Medicine, Weill Cornell Medical College, New York, NY;
  • Rita Shaknovich, 1Division of Hematology/Oncology, Department of Medicine, Weill Cornell Medical College, New York, NY;
  • Ling Wang, 1Division of Hematology/Oncology, Department of Medicine, Weill Cornell Medical College, New York, NY;
  • Shao Ning Yang, 1Division of Hematology/Oncology, Department of Medicine, Weill Cornell Medical College, New York, NY;
  • Kenny Ye, 3Division of Biostatistics, Department of Public Health, Albert Einstein College of Medicine, Bronx, NY;
  • Pedro Farinha, 4Department of Pathology, British Columbia Cancer Agency, Vancouver, BC; and
  • Douglas E. Horsman, 4Department of Pathology, British Columbia Cancer Agency, Vancouver, BC; and
  • Randy D. Gascoyne, 4Department of Pathology, British Columbia Cancer Agency, Vancouver, BC; and
  • Olivier Elemento, 5Institute for Computational Biomedicine, Weill Cornell Medical College, New York, NY
  • Ari Melnick, 1Division of Hematology/Oncology, Department of Medicine, Weill Cornell Medical College, New York, NY;

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